By American Academy of Ophthalmology, Ramana S. Moorthy MD
Starts off with an outline of immune-mediated eye ailment, summarizing simple immunologic strategies, ocular immune responses and distinct issues in ocular immunology. Discusses the scientific method of uveitis and studies noninfectious (autoimmune) and infectious kinds of uveitis, with an elevated part on viral uveitis and new fabric on infectious and noninfectious scleritis.
Enhanced detection of infectious brokers via immunologic and genetic equipment and biologic therapeutics are unique. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular facets of AIDS.
Upon of completion of part nine, readers could be capable to:
Identify normal and particular pathophysiologic approaches that impact the constitution and serve as of the uvea, lens, intraocular cavities, retina, and different tissues in acute and protracted intraocular inflammation
Differentiate and determine infectious and noninfectious uveitic entities
Choose acceptable exam ideas and appropriate ancillary experiences in response to even if an infectious or noninfectious reason is suspected
Read Online or Download 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis PDF
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Additional resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis
The secondary processing phase begins when these PPD-stimulated macrophages migrate into the draining lymph node, where they encounter memory T lymphocytes from the previous lung infection, leading to reactivation of memory T lymphocytes. The secondary effector phase commences when these reactivated memory T lymphocytes recirculate and home back into the dermis and encounter additional antigen and macrophages at the site, causing the T lymphocytes to become fully activated and release cytokines.
Fibrin dissolution is mediated by plasmin, which is activated from its zymogen precursor, plasminogen, by plasminogen activators such as tissue plasminogen activator. Thrombin, which is derived principally from platelet granules, is released after any vascular injury that causes platelet aggregation and release. Fibrin may be observed in severe anterior uveitis (the "plastic aqueous") . The role of fibrin deposition in the eye during uveitis is unknown, but it is thought to contribute to complications such as synechiae, cyclitic membranes, and tractional retinal detachment.
I ' ' / ( Cytoklnes ) ~ ' .... 2 "Resting" monocyte "Primed" macrophage ,4 ...... -, Scavenging Phagocytosis . ...... ~ Various Innate stimuli 3 ~- . ··········~........ "Stimulated" monocyte . ,.... c-yt-ok - ln_e__ s) ( Eicosanolds ) Wound repair Angiogenesis MIid Inflammation Schematic representation of macrophage activation pathway. Classically, resting monocytes are thought to be the principal noninflammatory scavenging phagocyte. 1, Upon exposure to low levels of interferon gamma (IFN-y) from T lymphocytes, monocytes become primed, up-regulating major histocompatibility complex (MHC) class II molecules and performing other functions .